MFV3D Book Archive > Nonfiction 9 > Download Core Concepts in Parenchymal Kidney Disease by David Philibert M.D., F.R.C.P.C. (auth.), Fernando C. PDF

Download Core Concepts in Parenchymal Kidney Disease by David Philibert M.D., F.R.C.P.C. (auth.), Fernando C. PDF

By David Philibert M.D., F.R.C.P.C. (auth.), Fernando C. Fervenza, Julie Lin, Sanjeev Sethi, Ajay K. Singh (eds.)

Core recommendations in Parenchymal Kidney Disease presents finished and cutting-edge info at the analysis, therapy, type and pathogenesis of glomerular and tubulointerstitial ailments. Chapters characteristic a variety of scientific situations and are authored by means of a workforce of well known specialists within the box. skilled clinicians and trainees alike will locate this authoritative connection with be a worthwhile source and contribution to the literature.

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Minimal-change glomerulopathy of adulthood. Am J Nephrol. 1988;8:291–7. 22. Tse KC, Lam MF, Yip PS, Li FK, Choy BY, Lai KN, et al. Idiopathic minimal change nephrotic syndrome in older adults: steroid responsiveness and pattern of relapses. Nephrol Dial Transplant. 2003; 18:1316–20. 23. Nolasco F, Cameron JS, Heywood EF, Hicks J, Ogg C, Williams DG. Adult-onset minimal change nephrotic syndrome: a long-term follow-up. Kidney Int. 1986;29:1215–23. 24. Fakhouri F, Bocquet N, Taupin P, Presne C, Gagnadoux MF, Landais P, et al.

From Shimada M, Araya C, Rivard C, Ishimoto T, Johnson RJ, and Garin EH. Minimal change disease: a “two-hit” podocyte immune disorder? Pediatr Nephrol. 2011;26(4): 645–49. With kind permission from Springer Science and Business Media in the liver and adipose tissue and previously had only been shown to have low-level renal expression as shown by Northern blot of whole kidney specimens [13]. Clement et al. demonstrated that Angptl4 expression is upregulated in the glomeruli of rats exposed to several models of podocyte injury, including puromycin [14].

Alternative methods to define genetic susceptibility in these cases are desirable. One complementary approach that has found success is the identification of genetic variants that, while present in unaffected members of the general population, nonetheless increases the risk of developing NS in those individuals that possess this variant. The R229Q variant of NPHS2 has a minor allele frequency of 2–7 % in healthy individuals of European descent [68]. Individuals with one or two copies of this allele do not seem to have increased risk of SRNS [69].

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