By Fouad K Habib
It has develop into transparent that medical discoveries and technological advances resulting in novel practices are taking place extra fast than they are often assessed by means of both medical trials or medical assessment. still, vital breakthroughs were made within the improvement of recent remedies with major implications for the administration of benign prostatic hyperplasia and prostate melanoma. delivering a broad medical heritage, The Prostate: New innovations and advancements covers subject matters starting from simple biology to the regulatory mechanisms that keep an eye on irregular progress and progre. Read more...
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Extra resources for Disorders of the Prostate : New Concepts and Developments
Zhou AM, Tewari PC, Bluestein BI, Caldwell GW, Larsen FL (1993) Multiple forms of prostate-specific antigen in serum: differences in immunorecognition by monoclonal and polyclonal assays [see comments]. Clin Chem 39:2483–2491. 02 Chapter 1194 27/4/04 9:27 am Page 21 2 Cellular and molecular mechanisms that modulate prostate cancer metastasis Christopher S Foster, Vasilliki Karavana, Youqiang Ke and Mustafa BA Djamgoz Introduction There is now little doubt that the majority of human cancers are caused by specific somatic mutations within the genome.
1998) Reference ranges for serum prostate-specific antigen (PSA) in Black and White men without cancer. Urology 52:967–973. Wennberg J, Gittelsohn A (1982) Variations in medical care among small areas. Sci Am 246:120–134. Williams AM, Simon I, Landis PK, et al. (1999) Prostatic growth rate determined from MRI data: age-related longitudinal changes. J Androl 20:474–480. Witjes WP, Aarnink RG, Ezz-el-Din K, et al. (1997) The correlation between prostate volume, transition zone volume, transition zone index and clinical and urody- namic investigations in patients with lower urinary tract symptoms.
The principle of this technique is that the genome of the original metastatic cells is partially cleaved using Hind III to yield DNA fragments in the range 500 kb to 5 mb. These fragments are then ligated with syn- thetic linkers that have been engineered to contain particular restriction enzyme cleavage sites. Thereafter, the original genomic DNA fragments derived from the partially cleaved genome may be identified and manipulated through the particular properties of the linking fragments. Since sites Not-1 and Sfi-1 are highly infrequent within the human genome (frequencies of the order 5ϫ10Ϫ5 bases and 5ϫ10Ϫ7 bases, respectively), subsequent use of these enzymes will release DNA fragments without appreciable damage to the intervening genes.