MFV3D Book Archive > Nonfiction 9 > Download Intensive Care Medicine: Annual Update 2009 by B. M. Tang, S. J. Huang, A. S. McLean (auth.), Prof. PDF

Download Intensive Care Medicine: Annual Update 2009 by B. M. Tang, S. J. Huang, A. S. McLean (auth.), Prof. PDF

By B. M. Tang, S. J. Huang, A. S. McLean (auth.), Prof. Jean-Louis Vincent (eds.)

The replace compiles the newest advancements in experimental and medical learn and perform in a single finished reference publication. The chapters are written through good famous specialists within the box of in depth care and emergency medication. it really is addressed to every body taken with inner drugs, anesthesia, surgical procedure, pediatrics, in depth care and emergency medicine.

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Kidney) [9] and different cells 1. Study level Trial design, clinical settings, treatment duration 2. Patient level Age, gender, disease severity 3. Organ level Heart, lung, kidney 4. Cellular level Neutrophils, lymphocytes, endothelium 5. Molecular level Receptors, cytokines, microbial products 6. Genomic level Polymorphisms, gene-expression, proteomics Fig. 1. The iceberg model to conceptualize the sources of heterogeneity in sepsis trials. Variables on the upper levels of the model are easier to discern and study.

Mitochondrial Biogenesis Mitochondrial biogenesis involves multiple transcriptional regulation pathways that require the expression of nuclear and mitochondrial genes. The number of mtDNA molecules doubles in every cell cycle, under normal physiological conditions. When 19 I 20 I A. Pyle, P. Chinnery, and S. Baudouin conditions change, the mtDNA copy number can be altered according to the energy need of the cell. Animal models of sepsis have demonstrated depletion in the number of heart [38] and liver [39] mitochondria.

1). The iceberg model provides a qualitative overview of the sources of heterogeneity. The complexity of the data increases progressively downwards in this model (Fig. 1). , liver vs. kidney) [9] and different cells 1. Study level Trial design, clinical settings, treatment duration 2. Patient level Age, gender, disease severity 3. Organ level Heart, lung, kidney 4. Cellular level Neutrophils, lymphocytes, endothelium 5. Molecular level Receptors, cytokines, microbial products 6. Genomic level Polymorphisms, gene-expression, proteomics Fig.

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